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Am J Physiol Lung Cell Mol Physiol 283: L766–L776, 2002. First published May 3, 2002; ajplung. ␥-Glutamyl transferase deficiency results in lung oxidant stress in normoxia JYH CHANG JEAN,1 YUE LIU,
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Document Date: 2014-07-26 20:25:35

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Ventura / Lincolnshire / Buckinghamshire / Boston / Billerica / Keene / StressGen Biotechnologies / Naugatuck / St. Louis / /

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IgG / Vector Laboratories / Wesco / Amersham / Improvision Open-Lab Users Software / Sigma Diagnostics / Intertech Resources / /

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Canada / United Kingdom / /

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FDA Phase / /

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Animal Science Center / University of Utah / Pulmonary Center / Emory University / laboratory of Dr. Robert Marc / University School / /

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oxidant gas exposure / g/ml solution / gas exchange surface / inhaled oxidant gas / stable products / Image processing / stable end product / /

MedicalCondition

cellular deficiency / vascular injury / injury / oxidant-induced injury / pulmonary edema / Epithelial cell injury / Genetic GGT deficiency / showed glutathione deficiency / Glutamyl transferase deficiency / severe bronchiolar cellular injury / glutamyl transaminase deficiency / GGT deficiency / hyperoxia revealed extensive airway epithelial cell injury / bronchiolar epithelial cell injury / absolute deficiency / vascular pulmonary edema / glutathione deficiency / deficiency / lung glutathione deficiency / /

Organization

Boston University School of Medicine / University of Utah / Salt Lake City / Department of Pediatrics / Laboratory and Animal Science Center / US Federal Reserve / Emory University / Atlanta / Pulmonary Center / American Physiological Society / Moran Eye Center / /

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Martin Joyce-Brady / Elizabeth Klings / Robert E. Marc / Lou Ann Brown / Yue Liu / /

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RT / /

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BAL / M-9 / GGTenu1 / /

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ELF / British Columbia / Victoria / Utah / Georgia / Massachusetts / /

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electrophoresis / antibodies / Biotechnology / Image processing / gene expression / genotype / /

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