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DNA repair / Tumor markers / Tumor suppressor genes / Breast cancer / Mutation / PARP1 / PARP inhibitor / BRCA1 / Base excision repair / Biology / Medicine / Oncology


Citation: Cell Death and Disease[removed], e1076; doi:[removed]cddis[removed] & 2014 Macmillan Publishers Limited All rights reserved[removed]www.nature.com/cddis Expression of base excision repair key factors and
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Document Date: 2014-02-20 10:01:36


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Moscow / Milano / Bari / San Giovanni Rotondo / St. Louis / Hilden / Boston / Hickson / Foster City / Adelaide / Chicago / Koukourakis / /

Company

SPSS Inc. / Life Technologies / Institutional Tissue Bank of NCRC / PARP1 / EuroClone SpA / Thomson / Sigma-Aldrich / Molecular Genetics Laboratory / NHEJ 1 Molecular Genetics Laboratory / Thermo Fisher Scientific Inc. / Qiagen / ADP / Clinical Pharmacology Laboratory / Khuder SA / PARP2 / Chakravarthy AB / Ford / BayesMendel Software / Ryder / IDC / Macmillan Publishers Limited / /

Country

Germany / Italy / United States / Monaco / /

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Event

Product Issues / /

Facility

Dana-Farber Cancer Institute / University of Bari / /

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IndustryTerm

active site / chemicals / genome surveillance machinery / statistical software / /

MarketIndex

Mib 1410 / /

MedicalCondition

BRCA1 dysfunction / sporadic breast cancers / BRCA2 mutant cancer / ovarian cancer / Double-strand break repair deficiency / human triple-negative breast cancer / non-small cell lung cancer / highly invasive breast tumor / breast tumor / Breast cancer / familial and sporadic breast cancer / adenocarcinoma / Tumor / familial breast cancers / sporadic breast cancer / triple-negative inflammatory breast cancer / cancer / tumors / homologous recombination deficiency / generally triple-negative breast cancer / carcinogenesis / BRCAX tumors / C et al. Targeting XRCC1 deficiency / resected non-small cell lung cancer / breast and ovarian cancer / invasive breast cancer / diseases / BRCA1- and BRCA2-related breast cancer / basal-like sporadic cancers / glioma cell lines / sporadic basal-like breast cancer / BRCA1and BRCA2-related breast cancer / Human cancers / carcinoma / J Cancer / colon cancer / D et al. BRCA1 dysfunction / non-small cell lung carcinomas / invasive lobular carcinoma / triple-negative breast cancer / MS / human breast cancer / pediatric rhabdomyosarcomas / germ cell tumors / familial breast cancer / breast cancers / triple-negative human breast carcinoma MDA-MB231 / triple-negative breast cancers / human adult gliomas / Disease / Cancer Cancer / human mammary adenocarcinoma cell lines / triple-positive sporadic cancers / HR deficiency / HR-deficient tumors / lung cancer / BRCA2-related breast cancers / Mol Cancer / /

MedicalTreatment

radiotherapy / radiation / chemotherapy / /

Organization

4Anatomopathology Unit / University of Bari / School of Medicine / 5Experimental Medical Oncology Unit / Fatah / Ministry of Health / 6Medical Genetics Unit / Dana-Farber Cancer Institute / National Cancer Research Centre / /

Person

Manipulation / Inhibition / Ann Surg Oncol / Caroline Oakley / Giovanni Paolo II / /

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Position

RT / pmiR-REPORT miRNA Expression Reporter / APEX1 reporter / *Corresponding author / Walker / Fisher / reporter / /

Product

cisplatin / Sigma / Alli / hydrocortisone / brca1 mutations / bases / environmental insults / cell lines / repair / genetic material / /

ProvinceOrState

British Columbia / South Dakota / /

RadioStation

Minisini AM / Russell AM / /

TVStation

Kyle / /

Technology

radiation / X-ray / Gene expression / chemotherapy / recombination / polymerization / directed mutagenesis / ATM / http / /

URL

www.ma.uni-heidelberg.de / www.nature.com/cddis / /

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