<--- Back to Details
First PageDocument Content
A549 cell / PI3K/AKT/mTOR pathway / Myc / Epithelial-mesenchymal transition / Phosphoinositide 3-kinase / AKT / Carcinogenesis / Non-small-cell lung carcinoma / T-cadherin / Biology / Medicine / Oncology
A549 cell
PI3K/AKT/mTOR pathway
Myc
Epithelial-mesenchymal transition
Phosphoinositide 3-kinase
AKT
Carcinogenesis
Non-small-cell lung carcinoma
T-cadherin
Biology
Medicine
Oncology

Alpha-enolase promotes cell glycolysis, growth, migration, and invasion in non-small cell lung cancer through FAK-mediated PI3K/AKT pathway

Add to Reading List

Source URL: www.jhoonline.org

Download Document from Source Website

File Size: 1,94 MB

Share Document on Facebook

Similar Documents

Axl is essential for VEGF-A-dependent activation of PI3K/Akt

Axl is essential for VEGF-A-dependent activation of PI3K/Akt

DocID: 1prZ0 - View Document

46 | P a g e  Review Article PI3K/AKT pathway in modulating glucose homeostasis and its alteration in Diabetes SOMAYEH ALSADAT HOSSEINI KHORAMI , ARIYO

46 | P a g e Review Article PI3K/AKT pathway in modulating glucose homeostasis and its alteration in Diabetes SOMAYEH ALSADAT HOSSEINI KHORAMI , ARIYO

DocID: 1p8fG - View Document

Combinatorial Testing of an ERK Inhibitor, SCHwith PI3K/AKT/mTOR Inhibitors in Melanoma Master Thesis In order to obtain the academic degree

Combinatorial Testing of an ERK Inhibitor, SCHwith PI3K/AKT/mTOR Inhibitors in Melanoma Master Thesis In order to obtain the academic degree

DocID: 19AFa - View Document

Alpha-enolase promotes cell glycolysis, growth, migration, and invasion in non-small cell lung cancer through FAK-mediated PI3K/AKT pathway

Alpha-enolase promotes cell glycolysis, growth, migration, and invasion in non-small cell lung cancer through FAK-mediated PI3K/AKT pathway

DocID: 17Kcn - View Document

OPEN  Citation: Cell Death and Disease[removed], e1391; doi:[removed]cddis[removed] & 2014 Macmillan Publishers Limited All rights reserved[removed]www.nature.com/cddis

OPEN Citation: Cell Death and Disease[removed], e1391; doi:[removed]cddis[removed] & 2014 Macmillan Publishers Limited All rights reserved[removed]www.nature.com/cddis

DocID: 101RK - View Document