<--- Back to Details
First PageDocument Content
Hypohidrotic ectodermal dysplasia / Ectodermal dysplasia / IKBKG / Point mutation / Tumor necrosis factor-alpha / Mutation / NF-κB / EDA / Genetic code / Biology / Genodermatoses / Cytokines
Date: 2014-08-26 09:45:12
Hypohidrotic ectodermal dysplasia
Ectodermal dysplasia
IKBKG
Point mutation
Tumor necrosis factor-alpha
Mutation
NF-κB
EDA
Genetic code
Biology
Genodermatoses
Cytokines

Microsoft Word - Manuscript-EDA.doc

Add to Reading List

Source URL: www.ias.ac.in

Download Document from Source Website

File Size: 850,81 KB

Share Document on Facebook

Similar Documents

Quantification of Cellular NEMO Content and Its Impact on NF-κB Activation by Genotoxic Stress

DocID: 1tRZA - View Document

Paper of the month, May 2014 Human procaspase-1 variants with decreased enzymatic activity are associated with febrile episodes and may contribute to inflammation via RIP2 and NF-κB signaling. Heymann MC, Winkler S, Luk

DocID: 1sbMx - View Document

Letter pubs.acs.org/ac Observing Single Cell NF-κB Dynamics under Stimulant Concentration Gradient Yousef Awwad,† Tao Geng,‡ Albert S. Baldwin,§ and Chang Lu*,†,¶

DocID: 1lpWa - View Document

Cell biology / Biology / Transcription factors / Programmed cell death / Biochemistry / Protein complexes / Cell signaling / NF-B / Tumor necrosis factor alpha / IB / CHUK / RELA

Supplementary Mathematical Methods The mathematical analysis of the NF-κB pathway regulation is discussed in two sections. First, we consider TNFα-TNFR1 interactions in the microfluidic chamber. Next, in a separate sin

DocID: 1lfms - View Document

Transcription factors / Cell signaling / Cell biology / Programmed cell death / NF-B / RELA / Tumor necrosis factor alpha / Apoptosis / IB / Fractional kill / Cell nucleus / Signal transduction

Single-cell variation leads to population invariance in NF-κB signaling dynamics Jacob J. Hughey1, Miriam V. Gutschow1, Bryce T. Bajar and Markus W. Covert* Department of Bioengineering Stanford University 443 Via Orteg

DocID: 1kKOw - View Document