both human disease / mast cell deficiency / neurologic disease / encephalomyelitis / experimental autoimmune encephalomyelitis / nonspecific and specific inflammatory processes / clinical disease / hind limb paralysis / acute septic peritonitis / inflammatory cytokines / multiple sclerosis / allergic inflammation / demyelination / similar disease / typical EAE disease / demyelinating diseases / early and severe disease / Severe Disease / MS / acute disease / forelimb paralysis / typical chronic experimental autoimmune encephalomyelitis / autoimmune diseases / dissociation / acute experimental autoimmune encephalomyelitis / paralysis / neurologic inflammation / bacterial infections / disease / significantly reduced disease / inflammatory demyelination / Pertussis / human multiple sclerosis / diabetes / Experimental Allergic Encephalomyelitis TNF / either EAE disease / allergy / less severe disease / inflammatory lesions / autoimmune demyelination / tumor necrosis / significantly less severe disease / neurologic disorders / autoimmune disease / diseases / human and murine disease / infection / subsequent EAE disease / inflammation / EAE disease / Experimental allergic encephalomyelitis / human disease / inflammatory / /
MedicalTreatment
immunization / adoptive transfer / immunotherapy / /
Organization
Institutional Animal Care and Use Committee / Department of Pathology and Graduate Program / National Multiple Sclerosis Society / National Center for Infectious Diseases / Department of Neurology / Leukemia Society of America / Centers for Disease Control and Prevention / Emory University / Emory University School of Medicine / Division of Parasitic Diseases / /
Person
Claire-Anne Gutekunst / Nat / Virginia H. Secor / T. Jin / Melissa A. Brown / W. Evan Secor / J. Immunol / / /
Position
WB / Fisher / /
Product
penicillin / estradiol / c-kit kinase / /
ProvinceOrState
Mississippi / Georgia / /
PublishedMedium
the Bonferroni post / The Journal of Experimental Medicine / Molecular Biology / /
Mast Cells Are Essential for Early Onset and Severe Disease in a Murine Model of Multiple Sclerosis By Virginia H. Secor,* W. Evan Secor,储 Claire-Anne Gutekunst,§ and Melissa A. Brown*‡ From the *Graduate Program in