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Proteins / Apoptosis / Programmed cell death / Non-homologous end joining / DNA-PKcs / H2AFX / Ataxia telangiectasia mutated / Homologous recombination / AKT / Biology / DNA repair / Cellular processes
Proteins
Apoptosis
Programmed cell death
Non-homologous end joining
DNA-PKcs
H2AFX
Ataxia telangiectasia mutated
Homologous recombination
AKT
Biology
DNA repair
Cellular processes

MutS homologue hMSH5: role in cisplatin-induced DNA damage response

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