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Gerontology / Chromosomes / Telomere / Non-homologous end joining / Aging / H2AFX / BRCA1 / Homology directed repair / Ageing / Biology / Genetics / DNA repair


53BP1 Regulates DSB Repair Using Rif1 to Control 5′ End Resection Michal Zimmermann et al. Science 339, [removed]); DOI: [removed]science[removed]This copy is for your personal, non-commercial use only.
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Document Date: 2013-04-16 18:07:47


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Tübingen / Potsdam / New York / Washington / DC / Brno / Monterotondo / /

Company

Metabolic Networks / 3European Molecular Biology Laboratory / X. Yu L. C. / HighWire Press / /

Country

Germany / Italy / United States / Czech Republic / /

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pence / /

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Event

Product Issues / /

Facility

Max Planck Institute / Skirball Institute / European Institute of Technology / Max Planck Institute of Molecular Plant Physiology / Masaryk University / terminal S/TQ / Rockefeller University / University School / /

IndustryTerm

live-cell imaging / online version / intricate genetic network / Online Material / /

MedicalCondition

induced telomere dysfunction / potential ataxia telangiectasia / Rif1 telomere dysfunction / cancer / 53BP1 deficiency / /

Organization

Faculty of Science / Department of Metabolic Networks / Department of Cell Biology / Max Planck Institute for Developmental Biology / Czech Science Foundation / New York University School of Medicine / Max Planck Institute of Molecular Plant Physiology / Rockefeller University / New York / Breast Cancer Research Foundation / American Association for the Advancement of Science / Institute of Technology / Laboratory for Cell Biology and Genetics / Masaryk University / Mouse Biology Unit / Skirball Institute / /

Person

Vanessa Wahl / Michal Zimmermann / /

Position

Cancer Society Research Professor / T.d. / /

Product

dexamethasone / the absence / cells / /

ProvinceOrState

New York / /

PublishedMedium

Molecular Biology / /

Technology

hybridization / ATM / html / UDP / DNA synthesis / recombination / /

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http /

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