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Neurology / Signal transduction / Cell signaling / Long-term depression / Presenilin / Long-term potentiation / Inositol trisphosphate / NMDA receptor / Ryanodine receptor / Biology / Neuroscience / Neurophysiology


ER Calcium and Alzheimer’s Disease: In a State of Flux Mark P. Mattson (23 March[removed]Science Signaling[removed]), pe10. [DOI: [removed]scisignal.3114pe10] The following resources related to this article are available on
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Document Date: 2011-04-11 12:11:04


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C terminus / National Institute / Laboratory of Neurosciences / N terminus / /

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online version / impaired mitochondrial energy production / article tools / imaging / neuronal networks / amyloidogenic processing / quantitative structural magnetic resonance imaging data / memory networks / reduced energy availability / cellular energy deficits / Recent in vivo calcium imaging / /

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early Alzheimer’s disease / neurological disorders / dysfunction / early-onset familial Alzheimer’s disease / excitotoxic necrosis / synaptic dysfunction / long-term depression / mitochondrial dysfunction / Alzheimer’s Disease / disease / diabetes / clinical depression / traumatic brain injury / mild cognitive impairment / familial Alzheimer’s disease / inflammation / /

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National Institute on Aging / Alzheimer’s Association / American Association for the Advancement of Science / Laboratory of Neurosciences / National Institute on Aging Intramural Research Program / /

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Mark P. Mattson Published / Nat / J. Physiol / M. S. Wolfe / J. Hardy / L. K. McEvoy / Mark P. Mattson / E. Tanzi / C. P. Holden / B. Wu / V / C. M. Fanger / A. D. Roses / E. M. Wijsman / A. Auffret / V / D. M. Rentz / /

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