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REVIEWS Expanding insights of mitochondrial dysfunction in Parkinson’s disease Patrick M. Abou-Sleiman, Miratul M. K. Muqit and Nicholas W. Wood
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Document Date: 2010-06-16 07:04:35


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Company

UPS / Pi / ADP / /

Country

United Kingdom / /

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Facility

Queen Square / Institute of Neurology / TIM complex / Greenamyre’s laboratory / /

IndustryTerm

mitochondrial processing peptidase / electron transport chain / phosphate / cleavage product / in vitro overexpression systems / energy / carrier frequency / cleavage site / cellular energy / model systems / treatment of the disease / /

MedicalCondition

mitochondrial injury / secondary mitochondrial dysfunction / chronic rotenone exposure / archetypical non-genetic disease / B-cell leukaemia / dysfunction / neurological dysfunction / further dysfunction / tremor / primary dysfunction / progressive external ophthalmoplegia / nigrostriatal dysfunction / injury / deafness / common neurodegenerative movement disorder / distinct clinical syndrome / bradykinesia and postural instability / probable disease / parkin dysfunction / mitochondrial dysfunction / PARK7 Parkinson’s disease / Alzheimer’s disease / human parkinsonism / I deficiency / cancer / disease / parkinsonism / neuroblastoma cell line overexpressing G309D PINK1 / tumour / PD-like syndrome / neuroblastoma cells / I deficiency A / Parkinson’s disease / proteasome system ROS Proteasome Proteasome dysfunction / nrn1868 Parkinson’s disease / deficiency / parkin deficiency / human neuroblastoma cell lines / /

MedicalTreatment

caloric restriction / /

Organization

Department of Molecular Neuroscience / ThiJ / Institute of Neurology / /

Person

Miratul M. K. Muqit / Patrick M. Abou-Sleiman / /

Position

Queen / /

Product

LG PC12 Radio / Ultrasone DJ1 Headphone/Headset / /

Technology

Neuroscience / tomography / siRNA / apoptosis / DNA Chip / /

URL

www.nature.com/reviews/neuro / /

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