Oslo / Palo Alto / New York / Heidelberg / Cleveland / New Haven / /
Company
K. P. White S. A. / European Molecular Biology Laboratory / R E C T I O N S A / M.N.A. / G. K. Chan S. A. / Cold Spring Harbor Laboratory / Cold Harbor Laboratory / /
Country
Germany / Norway / United States / /
Currency
pence / / /
Facility
University of Oregon / National Human Genome Research Institute / Stanford University / EST library / National Institute of Neurological Disorders / Hughes Medical Institute / University of Oslo / Berkeley Drosophila Genome Project/ Howard Hughes Medical Institute / Mad1/Mad2 complex / National Institute of General Medical Studes / /
National Human Genome Research Institute / National Institute of Health / University of Oregon / 4Howard Hughes Medical Institute / Department of Developmental Biology / Yale University School of Medicine / Department of Biological Sciences / Berkeley Drosophila Genome Project/ Howard Hughes Medical Institute / Department of Chemical Engineering / Department of Genetics / National Institute of Neurological Disorders and Stroke / Department of Biochemistry / National Institute of General Medical Studes / Stanford University / University of Oslo / /
Person
A. Michael Bate / Michelle N. Arbeitman / Eileen E. M. Furlong / J. L. DeRisi / V / Celiac Sprue / /
Position
second messenger / Medical Scientist / co-first author / /
Product
fs / soma / adenosine / /
ProvinceOrState
Oregon / New York / Connecticut / Montana / California / /
REPORTS pend on Hec1 may signal checkpoint activation through diffusible Mad2 complexes. In Hec1-depleted cells, this activity could be generated through CENP-E or BubR1. Because kinetochores were not stretched in Hec1-d