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 | Document Date: 2009-02-09 21:27:14Open Document File Size: 167,36 KBShare Result on FacebookCompanyMagosin SA / Fairchild / Ogden / Proc Natl Acad Sci U S A / Goldman / French SA / Shimizu / Stephens / Jebb SA / /CountryUnited States / United Kingdom / /Currencypence / USD / / /EventProduct Issues / /FacilityNational University of Singapore / terminal of NT / National University Hospital / The Children’s Medical Institute / /IndustryTermenergy regulation / obese gene product / normal energy homeostasis / constant energy intake / neural networks / energy homeostatic function / human energy homeostasis / tissue-specific posttranslational processing / tissue-specific post-translational processing / pharmaceutical / energy homeostasis / food / energy / impaired processing / energy balance / neural network / gene products / energy intake / increased food intake / neuroendocrine systems / proximal dibasic site / food intake / mechanism governing energy homeostasis / treatment of obesity / /MedicalConditionabsence seizures / pituitary dysfunction / Leptin Deficiency Leptin deficiency / novel single gene defects / severe earlyonset obesity / severe obesity / hyperinsulinemia / epidemic / cortisol deficiency / proopiomelanocortin deficiency / liver failure / pediatric-onset obesity / obesity epidemic / dysfunction / B (TrkB) deficiency / severe early-onset obesity / Prader-Willi Syndrome / genetic mutations causing obesity / Licinio J. Human leptin deficiency / starvation / early onset obesity / complex / multifactorial disorder / obesity / Three leptin deficiency / disorder / leptin receptor deficiency / hypogonadism / monogenic obesity / childhood obesity / delayed puberty / MS / Receptor deficiency MC3R / severe glucocorticoid deficiency / mouse diabetes / hypercortisolism / precocious puberty / leptin deficiency / extreme early onset obesity / receptor deficiency / early onset severe obesity / neuroendocrine/metabolic dysfunction / Rahilly S. Monogenic human obesity / congenital leptin deficiency / human obesity / unique metabolic syndrome / Pro-opiomelanocortin Deficiency The pro-opiomelanocortin / genetic disorder / hypothermia / diabetes / congenital deficiency / genes governing obesity / Genetically transmitted obesity / namely leptin deficiency / Obesity Introduction Obesity / high fat diet-induced obesity / immune system dysfunction / extreme obesity / human congenital leptin deficiency / Partial leptin deficiency / hypogonadotropic hypogonadism / subclinical hypothyroidism / Multi-factorial Trait Obesity / POMC deficiency / developed obesity / deficiency / MC4R deficiency / Bouchard C. Obesity / Common obesity / cholestasis / syndrome / ACTH deficiency / morbid obesity / /OrganizationNational University of Singapore / Children’s Medical Institute / National University Hospital / Annals Academy of Medicine Leptin-melanocortin and Weight Regulation / Department of Paediatrics / /PersonLee Farooqi / Yung Seng Lee / Ann Acad Med / Connie Hung / Lee Yung Seng / /PositionButler / messenger / important distal mediator / /Productchorionic gonadotropin / ACTH / intracellular transport / mediators / leptin receptors / /ProgrammingLanguageML / /RadioStationDePaoli AM / /Regionnorthern Algeria / /TechnologyGenomics / antibodies / hybridization / Positional cloning / cloning / neural network / gene expression / genotype / /SocialTag |