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City Phoenix / TRF2DBDM -expressing / TRF2DBDM-expressing / Santa Cruz / / Company Singer / Cox / LinkeS.P. / FosterS.A. / Becton Dickinson and Co. / anti-TRF2 Ab / Amersham / Novocastra Laboratories / Titia de Lange1 Laboratory / / / Event FDA Phase / Product Issues / / Facility G1 checkpoint / Lange laboratory / The Rockefeller University / / IndustryTerm test systems / p19Arf product / fusion products / 3T3 protocol / / MedicalCondition cancers / tumor / T.de Lange dysfunction / cancer / tumors / carcinogenesis / weak tumor / dysfunction / mouse tumor / intestinal carcinomas / telomere dysfunction / human tumors / telomere tumor / MEFs experiencing TRF2DBDMmediated telomere dysfunction / incipient tumor / p53±/± tumor / human telomere tumor / acute telomere dysfunction / skin tumors / infection / p53/RB/telomere/TRF2/tumor suppressor Introduction In / potential tumor / established tumor / control virus infection / / Organization National Institute of Health / Cornell / Rockefeller University / / Person Agata Smogorzewska / Nat / McKnight / Scott Lowe / / Position author / G.P. / T.D. / player / Harper / / Product penicillin / Sigma / glycine / p53 pathway / 3T3 / / ProvinceOrState A.B. / / Technology 3T3 protocol / antibodies / Biotechnology / hybridization / gene delivery / ATM / DNA binding / 3G / gel electrophoresis / apoptosis / /
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The EMBO Journal Vol. 21 No. 16 pp. 4338±4348, 2002 Different telomere damage signaling pathways in human and mouse cells Agata Smogorzewska and Titia de Lange1