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REPORTS expression is responsible for the cre1 ahk3 phenotype, because loss of AHP6 function in ahp6-1 cre1 ahk3 was able to suppress the ectopic protoxylem (91%; n[removed]Fig. 1B). In both wol and cre1 ahk2 ahk3, the AH
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Document Date: 2012-05-16 08:34:46
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