| Document Date: 2014-09-25 08:35:59
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City Mainz / Heidelberg / / Company Macmillan Publishers Limited / / Country Germany / / / Facility Johannes Gutenberg University / Institute of Molecular Cell Biology / / / IndustryTerm chemical chaperone / sphingolipid metabolic/catabolic network / cancer therapy / / MedicalCondition Program Infection / tumor / different tumor / cancer / tumors / Disease / increased tumor / human glioblastoma cell lines / glioblastoma cell death / Glioblastomas / Glioblastoma / / MedicalTreatment cancer therapy / / Organization German Cancer Research Centre / 4German Cancer Research Centre / Institute of Molecular Cell Biology / / / Position representative / Germany *Corresponding author / / Product etoposide / / ProvinceOrState Saskatchewan / / Technology treating cancer / siRNA / apoptosis / atg / / URL www.nature.com/cddis / /
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A sphingosine kinase inhibitor combined with temozolomide induces glioblastoma cell death through accumulation of dihydrosphingosine and dihydroceramide, endoplasmic reticulum stress and autophagy