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Apoptosis / Immune system / Signal transduction / Protein families / Aryl hydrocarbon receptor / Fas receptor / Caspase / Tumor necrosis factor-alpha / Poly ADP ribose polymerase / Biology / Cell biology / Programmed cell death


Activation of the aryl hydrocarbon receptor sensitises human keratinocytes for CD95L- and TRAIL-induced apoptosis
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University Hospital Wuerzburg / Midwest Research Institute / German Federal Institute / Heinrich Heine University / stable k.d. / ng/ml TRAIL / /

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subsequent processing / cleavage products / equivalent processing pattern / image processing program / Chemical carcinogenesis / chemical inhibition / stock solutions / chemical exposures / chemicals / /

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secondary genotoxic injuries / metastasis / J Cancer / toxic epidermal necrolysis / Stevens-Johnson syndrome / inflammatory molecules / inflammatory skin lesions / eczematous dermatitis / MS / inflammatory diseases / Chemical carcinogenesis / colon carcinoma / mitochondrial dysfunction / human p53 wild-type cancer / Disease / tumour necrosis factor-a / cellular injuries / human lung cancer / tumours in mouse skin / tumour / inflammatory skin diseases / Tumor necrosis / lung cancer / rare skin disease / inflammation / inflammatory / /

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radiation / /

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Heinrich Heine University / Department of Product Safety / Midwest Research Institute / Federal Ministry of Education / German Federal Institute for Risk Assessment / /

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Arch Biochem Biophys / /

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mediator / Germany *Corresponding author / /

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penicillin / growth factor / sterile water / hydrocortisone / ahr / synthetic materials / CYP1B1 / HaCaT variants / cells / materials / /

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Java / /

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South Dakota / /

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Westendorf AM / /

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radiation / antibodies / siRNA / Biotechnology / apoptosis / Cell Signaling Technology / /

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