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Tumor necrosis factor-alpha / TRADD / Apoptosis / FADD / Caspase 3 / Caspase / Fas receptor / Death effector domain / Tumor necrosis factors / Biology / Cell biology / Programmed cell death


TNF can activate RIPK3 and cause programmed necrosis in the absence of RIPK1
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Document Date: 2013-01-17 09:59:28

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Research Institute / University of Cologne / I complex / Imperial College / University of California San Diego / Osaka University / Micromon DNA Sequencing Facility / University of Massachusetts Medical School / La Trobe Institute / Eliza Hall Institute / La Trobe University / Eliza Hall Institute of Medical Research / RIP3 complex / The University of Melbourne / University of Tasmania / /

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TNF-induced necrosis / JE / virus-induced programmed necrosis / murine liver injury / programmed necrosis / necrosis / lentiviral infections / Disease / virus-induced inflammation / neuroblastoma cells / p55 tumor necrosis / tumor necrosis / tumor necrosis factor / infection / /

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University of Melbourne / Research Institute for Microbial Diseases / La Trobe Institute for Molecular Science / University of Cologne / Cologne / Imperial College / La Trobe University / Osaka University / Department of Biochemistry / African Union / Eliza Hall Institute / University of California San Diego / San Diego / Eliza Hall Institute of Medical Research / University of Massachusetts Medical School / University of Tasmania / /

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Victoria / Tasmania / Massachusetts / /

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alpha / antibodies / apoptosis / DNA Sequencing / 3G / /

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