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Intensive care medicine / Immune system / Autophagy / Cytokines / Sepsis / Lipopolysaccharide / Toll-like receptor / Multiple organ dysfunction syndrome / Tumor necrosis factor-alpha / Biology / Cell biology / Programmed cell death


Peroxisome proliferator-activated receptor α activation attenuates the inflammatory response to protect the liver from acute failure by promoting the autophagy pathway
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Document Date: 2014-08-28 08:36:35

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File Size: 3,07 MB

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Dallongeville / Shanghai / Leitersdorf / Helsinki / Tokyo / Chengdu / Suzhou / Cambridge / Beijing / Shijiazhuang / San Diego / St. Louis / Santa Cruz / /

Company

ALF / IgG / Cell Signaling Technology Inc. / Nikon Corp. / Thermo Fisher Scientific / Sigma-Aldrich / Engreen Biosystem Co. / Bio-Rad / Macmillan Publishers Limited / /

Country

Japan / United States / China / /

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Event

Product Issues / Extinction / /

Facility

Training Center / The Third Affiliated Hospital of Hebei Medical University / Biochain Institute / Capital Medical University / Institute of Hepatology / Beijing YouAn Hospital / The College of Life Sciences / /

IndustryTerm

study protocol / experimental / therapy for the end stage of the disease / normal saline solution / macrophage-conditioned media / /

MedicalCondition

inflammatory response / inflammatory reaction / liver inflammation / reperfusion injury / inflammatory processes / HBV / massive hepatic injury / inflammatory signaling / suppressed hepatic inflammation / hepatitis B / inflammatory mechanisms / inflammatory cytokines / injury / Ascher N. Acute hepatic failure / clinical syndrome / D-GalN/LPS-induced liver injury / end-stage liver disease / encephalopathy score Child / HBV infection undergoing liver transplantation / breast cancer / sepsis / inflammatory stimuli / tumor necrosis factor-a / hepatic inflammation / acute liver failure / indirectly alleviating inflammation / systemic inflammatory response / HBV infection / Disease / hepatic inflammatory injury / hepatocyte injury / tumor necrosis / infection / viral hepatitis / inflammatory pathways / liver injury / inflammation / STAT1 inflammatory signaling / chronic hepatitis B / inflammatory responses / hepatocellular injury / /

Organization

Capital Medical University / Beijing / Beijing YouAn Hospital / Kota BP / College of Life Sciences / 2Beijing Artificial Liver Treatment and Training Center / Ministry of Personnel of Beijing / Third Affiliated Hospital / Hebei Medical University / National Natural Science Foundation of China / Duan / Beijing Artificial Liver Treatment and Training Center / 3Beijing Institute of Hepatology / Technology Foundation / Biochain Institute / Capital Medical University Animal Care Committee / 5Sichuan University / Medical Ethics Committee / Department of Infectious Diseases / /

Person

Alexa Fluor / Williams R. Mechanisms / /

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Position

General / Lamp1 Forward / Representative / /

Product

PBS injection / penicillin / Sigma / IL-6 / IL-1b / D-GalN/LPS injection / D-GalN/ LPS injection / IL-12p40 iNOS / organelles / /

Technology

study protocol / Antibodies / siRNA / transplantation / apoptosis / Gene expression / 3g / gel electrophoresis / /

URL

www.nature.com/cddis / /

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