Back to Results
First PageMeta Content
Cell signaling / Apoptosis / Immune system / Signal transduction / Proteins / NF-κB / Tumor necrosis factor-alpha / Apoptosome / Bcl-2-associated X protein / Biology / Cell biology / Programmed cell death


An upper limit for macromolecular crowding effects
Add to Reading List

Open Document

File Size: 1,64 MB

Share Result on Facebook

City

Shanghai / Kunzendorf / Shiga / /

Company

SPSS Inc. / China 2 Shanghai Key Laboratory / Amersham / Ligand / Google / Hitachi / Takara Bio Inc. / Sanz AB / BioMed Central Ltd. / Bio-Rad / Suzuki / MacMillan / performed using SPSS / Creative Commons / /

Country

Taiwan / Japan / China / /

/

Event

Environmental Issue / Reorganization / /

Facility

Zhongshan Hospital / Shanghai Key Laboratory of Organ Transplantation / Fudan University Zhongshan Hospital / IRAK complex / Fudan University / /

IndustryTerm

treatment of infectious and inflammatory diseases / food / amplification protocol / innate and adaptive immune systems / oxidation products / breakdown products / terminal product / online submission / /

MedicalCondition

inflammatory response / nonantigen-dependent inflammatory pathways / reperfusion injury / acute kidney failure / subsequent inflammatory responses / ischemic neuronal injury / Ischemic Acute Kidney Injury / endothelial injury / Tissue injury / cerebral ischemia/reperfusion injury / polymicrobial sepsis / kidney ischemia/ reperfusion injury / tissue injuries / Subsequent mitochondrial dysfunction / ischemia/ reperfusion injury / ischemia/reperfusion-induced renal injury / Apoptotic Ischemia-Reperfusion Injury / renal dysfunction / inflammatory diseases / hypoxia/ reoxygenation injury / Acute Ischemia-reperfusion Injury / Programmed necrosis / inflammatory models / mitochondrial dysfunction / experimental bacterial meningitis / Ischemia/Reperfusion Injury / renal inflammation / postischemic reperfusion injury / Apoptosis Background Renal ischemia-reperfusion injury / infectious and inflammatory diseases / immune-mediated injury / acute kidney injury / histological injury / active immune-mediated injury / tubulointerstitial inflammation / long-term renal ischemia/reperfusion injury / mastitis / Renal injuries / inflammatory cytokine genes / ischemia-reperfusion injury / antigen-independent inflammatory process / IRI-induced mitochondrial dysfunction / important inflammatory cascade / renal ischemia-reperfusion injury / renal ischemia/reperfusion injury / inflammation / inflammatory / /

MedicalTreatment

incision / kidney transplants / kidney transplantation / surgery / /

Organization

Bioethics Committee / Department of Urology / Science and Technology Commission / Zhongshan Hospital / Fudan University / Shanghai / US Federal Reserve / Fudan University Zhongshan Hospital / Ministry of Health / National Nature Science Foundation of China / /

Person

Exp Biol Med / Chin Med / Van Schadewijk / Arch Biochem Biophys / Grant Nos / /

Position

Cao / Yin HB / /

Product

pentobarbital sodium / Sigma / DV / /

ProgrammingLanguage

ML / /

ProvinceOrState

Shanghai Municipality / /

PublishedMedium

PLoS ONE / /

RadioStation

Ramos AM / /

Technology

alpha / radiation / amplification protocol / antibodies / transplantation / apoptosis / Cell Signaling Technology / /

URL

www.biomedcentral.com/submit / http /

SocialTag